Evaluation of Left Ventricular Circumferential Contraction Functions in Obese Patients

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dc.contributor.authorSurucu, Huseyin
dc.contributor.authorTatli, Ersan
dc.contributor.authorBoz, Hakki
dc.contributor.authorMeric, Mehmet
dc.date.accessioned2024-06-12T11:07:36Z
dc.date.available2024-06-12T11:07:36Z
dc.date.issued2010
dc.departmentTrakya Üniversitesien_US
dc.description.abstractBackground: We aim to evaluate left ventricular (LV) function abnormalities, especially circumferential contraction functions, in obese patients. Method: Cases without coronary artery disease (CAD) were divided into two groups according to their body mass indexes (BMI). Results: Female predominance (P = 0.002), systolic blood pressure (BP) (P = 0.001), diastolic BP (P = 0.001), waist circumference (P < 0.001), left atrium (P < 0.001), LV end-diastolic diameter (P = 0.046), LV mass index (P = 0.001), and LV stroke volume (P = 0.016) were prominent in obese patients (BMI >= 27). In obese patients, transmitral late velocity (P = 0.005) was prominent, and pulmonary vein antegrade diastolic velocity (PV-D) (P = 0.002) and mitral annular early diastolic pulsed-wave tissue Doppler imaging (pw-TDI) velocity (annular Ea) (P = 0.032) were lower. Transmitral late velocity was positively correlate with stroke volume (P = 0.029) and systolic BP (P < 0.001). Negatively correlation between PV-D and diastolic BP (P = 0.046) was found. And also, annular Ea velocity was negatively correlate with systolic BP (P = 0.017) and diastolic BP (P = 0.031). These findings may reflect LV longitudinal contraction abnormalities (LVLCA) and underlying mechanism that is responsible for LVLCA, may be volume and afterload alterations. However, LV circumferential contraction functions that evaluate by using pw-TDI, were not different among the groups. Conclusion: In obese patients without CAD, it was clearly said that while LVLCA were evident, LV circumferential contraction abnormalities were not. This differentiation may be explained by subepicardial myocardial fiber that is responsible for LV circumferential contractions is supplied by coronary arteries, subendocardial myocardial fiber that is responsible for LV longitudinal contractions, is supplied by systemic circulation via LV cavity penetration. (ECHOCARDIOGRAPHY 2010;27:378-383).en_US
dc.identifier.doi10.1111/j.1540-8175.2009.01036.x
dc.identifier.endpage383en_US
dc.identifier.issn0742-2822
dc.identifier.issue4en_US
dc.identifier.pmid20113326en_US
dc.identifier.scopus2-s2.0-77951232903en_US
dc.identifier.scopusqualityQ3en_US
dc.identifier.startpage378en_US
dc.identifier.urihttps://doi.org/10.1111/j.1540-8175.2009.01036.x
dc.identifier.urihttps://hdl.handle.net/20.500.14551/22093
dc.identifier.volume27en_US
dc.identifier.wosWOS:000276855700007en_US
dc.identifier.wosqualityQ3en_US
dc.indekslendigikaynakWeb of Scienceen_US
dc.indekslendigikaynakScopusen_US
dc.indekslendigikaynakPubMeden_US
dc.language.isoenen_US
dc.publisherWiley-Blackwellen_US
dc.relation.ispartofEchocardiography-A Journal Of Cardiovascular Ultrasound And Allied Techniquesen_US
dc.relation.publicationcategoryMakale - Uluslararası Hakemli Dergi - Kurum Öğretim Elemanıen_US
dc.rightsinfo:eu-repo/semantics/closedAccessen_US
dc.subjectLeft Ventricular Circumferential Contractionen_US
dc.subjectObesityen_US
dc.subjectPulsed-Wave Tissue Doppler Imagingen_US
dc.subjectMorbid-Obesityen_US
dc.subjectWeight-Lossen_US
dc.subjectEchocardiographyen_US
dc.subjectHypertensionen_US
dc.subjectHypertrophyen_US
dc.subjectAxisen_US
dc.subjectCardiomyopathyen_US
dc.subjectVolumeen_US
dc.subjectWomenen_US
dc.subjectMassen_US
dc.titleEvaluation of Left Ventricular Circumferential Contraction Functions in Obese Patientsen_US
dc.typeArticleen_US

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