Movement Disorder Due to Involvement of Bilateral Basal Ganglia in Diabetic Uremic Patient
dc.contributor.author | Çakar, Merve Melodi | |
dc.contributor.author | Özkan, Hülya | |
dc.date.accessioned | 2024-06-12T10:05:30Z | |
dc.date.available | 2024-06-12T10:05:30Z | |
dc.date.issued | 2020 | |
dc.department | Trakya Üniversitesi | en_US |
dc.description.abstract | Basal ganglia, which are one of the most active and permeable regions of the brain, can be reversibly or irreversibly damaged due to metabolic diseases, carbon monoxide intoxication, hypoxia, infectious and vasculitic causes. This may lead to various manifestations, especially movement disorders, but also epilepsy, encephalopathy and gait abnormalities. We present a 64-year-old female patient who had involuntary movements of the limbs. Her diffusion brain magnetic resonance imaging (MRI) showed bilateral basal ganglia hyperintense le-sions in T2 weighted flair sequences. The involvement of the basal ganglia is explained by lactic acidosis, caused by metformin usage despite chronic renal failure. In this study, we present a case of generalized choreatethosis and involvement of basal ganglia due to lactic acidosis in end stage renal failure. | en_US |
dc.identifier.doi | 10.5336/caserep.2019-71948 | |
dc.identifier.endpage | 195 | en_US |
dc.identifier.issn | 2147-9291 | |
dc.identifier.issue | 3 | en_US |
dc.identifier.startpage | 192 | en_US |
dc.identifier.trdizinid | 410098 | en_US] |
dc.identifier.uri | https://doi.org/10.5336/caserep.2019-71948 | |
dc.identifier.uri | https://search.trdizin.gov.tr/yayin/detay/410098 | |
dc.identifier.uri | https://hdl.handle.net/20.500.14551/13486 | |
dc.identifier.volume | 28 | en_US |
dc.indekslendigikaynak | TR-Dizin | en_US |
dc.language.iso | en | en_US |
dc.relation.ispartof | Türkiye Klinikleri Journal of Case Reports | en_US |
dc.relation.publicationcategory | Makale - Ulusal Hakemli Dergi - Kurum Öğretim Elemanı | en_US |
dc.rights | info:eu-repo/semantics/openAccess | en_US |
dc.title | Movement Disorder Due to Involvement of Bilateral Basal Ganglia in Diabetic Uremic Patient | en_US |
dc.type | Article | en_US |