Type 2 diabetes mellitus

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dc.authorscopusid7004461946
dc.contributor.authorTu?rul A.
dc.date.accessioned2024-06-12T10:28:19Z
dc.date.available2024-06-12T10:28:19Z
dc.date.issued2007
dc.description.abstractType 2 diabetes mellitus comprises a group of common metabolic disorders that the phenotype of hyperglycaemia. Hyperglycaemia may include reduced insulin secretion, decreased glucose utilization and increased glucose production. The metabolic dysreguiation associated with type 2 diabetes mellitus causes secondary pathophysiologic changes in multipl organ systems. Glycemic control decreases the risk of diabetes complications.en_US
dc.identifier.endpage93en_US
dc.identifier.issn1016-5134
dc.identifier.issue6en_US
dc.identifier.scopus2-s2.0-34447636946en_US
dc.identifier.scopusqualityN/Aen_US
dc.identifier.startpage88en_US
dc.identifier.urihttps://hdl.handle.net/20.500.14551/17163
dc.identifier.volume19en_US
dc.indekslendigikaynakScopusen_US
dc.language.isotren_US
dc.relation.ispartofSENDROMen_US
dc.relation.publicationcategoryMakale - Uluslararası Hakemli Dergi - Kurum Öğretim Elemanıen_US
dc.rightsinfo:eu-repo/semantics/closedAccessen_US
dc.subject2,4 Thiazolidinedione Derivative; Acarbose; Acetylsalicylic Acid; Angiotensin Receptor Antagonist; Dipeptidyl Carboxypeptidase Inhibitor; Insulin; Metformin; Sulfonylurea; Article; Atherosclerosis; Diabetic Nephropathy; Gluconeogenesis; Glucose Blood Level; Glucose Utilization; Glycemic Control; Human; Hyperglycemia; Insulin Release; Non Insulin Dependent Diabetes Mellitus; Pathophysiology; Phenotypeen_US
dc.titleType 2 diabetes mellitusen_US
dc.title.alternativeTip 2 diabetes mellitusen_US
dc.typeArticleen_US

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