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Öğe Öğe Electroclinical patterns in patients with nonconvulsive status epilepticus: Etiology, treatment, and outcome(Academic Press Inc., 2021) Baysal-Kirac L.; Cakar M.M.; Altiokka-Uzun G.; Guncan Z.; Guldiken B.Introduction: This study investigated the clinical and electroencephalography (EEG) features and prognostic factors of patients with nonconvulsive status epilepticus (NCSE). Materials and methods: We retrospectively reviewed the clinical files and EEG data of 45 (28 females, mean age 54 ± 22.6 years) consecutive patients with NCSE over a five-year period. An EEG interpreter who was blinded to the clinical findings evaluated the EEGs according to the Salzburg Consensus Criteria (SCC) for NCSE. Patient demographics, etiology, neuroimaging and laboratory data, EEG features, treatment, and outcome measures were analyzed. Results: The most common etiology for NCSE was acute symptomatic etiologies (57.8%) and cerebrovascular disease (48.9%). The majority (68.9%) of the patients presented with new-onset status epilepticus (SE). NCSE was refractory to treatment in 31.1% of patients. The most common status pattern consisted of rhythmic delta/theta activity in 62.3% of EEGs. Twenty-five status patterns on the EEGs were classified as definite, 30 as possible, and six as no NCSE according to the SCC. The in-hospital mortality rate was high (33.3%) showing an association with potentially fatal etiology, refractory SE, treatment with continuous I.V. anesthetics and also the presence of multiple status patterns and nonreactivity in EEGs (p < 0.05). Conclusions: The SCC for NCSE have high diagnostic accuracy but do not affect prognosis. Potentially fatal etiology, multiple status patterns on EEG and non-reactive EEGs may carry significantly greater risk for short-term mortality. © 2020 Elsevier Inc.Öğe Electrophysiological risk factor in the development of diabetic foot: Pobterior tibial nerve conduction pathology(2009) Taşkiran B.; Güldiken S.; Turgut N.; Guldiken B.; Tu?rul A.Objective: Vascular insufficiency, polyneuropathy, infection, and pressure are among the multiple factors involved in the development of diabetic foot as a delayed complication of diabetes mellitus. In this study we aimed to determine the leading nerve conduction pathologies operating in diabetic lesions of the lower extremities. Method: In this study, 20 diabetic patients with diabetic foot and polyneuropathy, 20 patients with only polyneuropathy, and 20 diabetics without any of the stated complications were evaluated retrospectively. Age, duration of diabetes, haemoglobin A1c, and electroneuromyography findings of these three groups were compared. Results: Patients with diabetic foot were older and had longer duration of diabetes and higher levels of haemoglobin A1c. Peroneal sensorimotor nerve conduction studies did not differ between the patients with diabetic foot and those with polyneuropathy but without diabetic foot. Posterior tibial motor nerve conduction velocity and amplitude were significantly lower in the first group (p<0.05). Posterior tibial motor nerve conduction velocity was inversely related to the duration of diabetes (p<0.01, r= -0.9). There was no relation with other daemographic features and metabolic values. Discussion: Decreases in posterior tibial motor nerve conduction velocity and M-response amplitude are risk factors for diabetic foot. We are the opinion that patients with ENMG eliciting such results should be followed up more carefully.
