Yazar "Esgin, H." seçeneğine göre listele

Listeleniyor 1 - 3 / 3
  • Küçük Resim Yok
    Öğe
    Effect of oxygenation and suction in monitored anaesthesia care of eye surgery
    (Lippincott Williams & Wilkins, 2004) Turan, A.; Esgin, H.; Seker, S.; Pamukcu, Z.
    [Abstract Not Available]
  • Küçük Resim Yok
    Öğe
    Functional assessment of reattached macula in nine cases with excellent Snellen acuities
    (Nature Publishing Group, 2007) Ozgur, S.; Esgin, H.
    Purpose To compare the macular functions of patients with excellent visual acuity after retinal detachment (RD) surgery, with the healthy fellow eye. Methods Of 214 patients, nine patients, who were successfully operated because of unilateral RD involving the macula and achieved excellent visual acuity, were analysed. The fellow eyes of the patients were taken as the control group. The macular functions were evaluated with visual acuity, contrast sensitivity, colour vision, visual field, and pattern VEP in operated and nonoperated eyes. Results There was no significant difference in visual acuity, contrast sensitivity, colour vision, and VEP outcomes between operated and nonoperated eyes. However, it was seen that the mean deviation in the visual field was significantly higher in the operated eyes (5.87 +/- 1.8dB) when compared with the undetached fellow eyes (3.1 +/- 1.8 dB), (t = 12.5; P = 0.013). Conclusion Even though the visual acuity, colour vision, contrast sensitivity, and VEP results returned to normal after a successful RD surgery, we found that the mean deviation measured by the visual field, which reflects the retinal sensitivity, was still significantly low after nearly 5 years follow-up.
  • Küçük Resim Yok
    Öğe
    VON HIPPEL-LINDAU DISEASE: THE CLINICAL MANIFESTATIONS AND GENETIC ANALYSIS RESULTS OF TWO CASES FROM A SINGLE FAMILY
    (Macedonian Acad Sciences Arts, 2015) Kinyas, S.; Ozal, S. A.; Guclu, H.; Gurlu, V; Esgin, H.; Gurkan, H.
    von Hippel-Lindau (VHL) disease is an autosomal dominant inherited multi systemic cancer syndrome that is classically associated with neoplasms in multiple organs, and caused by mutations in the VHL gene on chromosome 3p25-p26. Retinal hemangioblastoma (RH) is the most frequent and the earliest clinical sign of the disease, which is seen in 40.0-60.0% of patients. In recent years, studies of patients with VHL tried to put forward the relationship between genotype and phenotype. In this study, two VHL cases in the same family with clinical findings and genetic analysis results are presented. As a consequence of the genetic studies, a heterozygous missense mutation c.202 T>C, p.S68P (Ser68Pro) in exon 1 of the VHL gene that is mapped to chromosome 3p25.3, was found in the patients' DNA sample. The germline mutation of [c.202T>C, p.S68P (Ser68Pro)] that was detected in both cases, has been reported in only two cases in the literature. However, in these reported cases, any systemic involvement except RH, were not reported. Although our cases had the same mutation, we detected renal involvement in both cases, and also central nervous system (CNS) involvement in one case, in addition to RH.